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glial scar formation  (NeuroRepair)

 
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    NeuroRepair glial scar formation
    Glial Scar Formation, supplied by NeuroRepair, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/glial+scar+formation/glial+scar+formation/pm29727731-111-4-17
    Average 90 stars, based on 1 article reviews
    glial scar formation - by Bioz Stars, 2026-10
    90/100 stars

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    Related Articles

    Control:

    Article Title: Effects of long-term rapamycin treatment on glial scar formation after cryogenic traumatic brain injury in mice.
    Article Snippet: Glial scar impedes axon regeneration and functional recovery following traumatic brain injury (TBI).. Although it has been shown that rapamycin (a specific inhibitor of mammalian target of rapamycin) can reduce astrocyte reactivation in the early stage of TBI, its effect on glial scar formation has not been characterized in TBI and other acute brain injury models.. To test this, ICR mice received daily administration of rapamycin (0.5 or 1.5 mg/kg, i.p.) beginning at 1 h after cryogenic TBI (cTBI).



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    The combined treatment of TUDCA and BMSC transplantation accelerated the regeneration of tissues and motor functional recovery after SCI. a Images of behavioral observation showing the characteristics of hind limb movement of rats across different groups. b Footprint analyses of different groups on day 28 after SCI. c Representative images from H&E staining in longitudinal section on day 28 after SCI. d , e The survived neurons following Nissl staining and relative quantification at the lesion area from different groups on day 28 after SCI. f The Basso, Beattie, and Bresnahan (BBB) locomotor scores of different groups. g The Immunofluorescence staining <t>of</t> <t>MAP2(green),</t> MBP (green), TUJ1 (red), and <t>GFAP</t> (red) from different groups on day 28 post-SCI. Data are presented as mean ± SD. * P < 0.05 vs. the SHAM group. # P < 0:05 vs. the SCI group. & P < 0.05 vs. the BMSC group ( n = 6)
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    The combined treatment of TUDCA and BMSC transplantation accelerated the regeneration of tissues and motor functional recovery after SCI. a Images of behavioral observation showing the characteristics of hind limb movement of rats across different groups. b Footprint analyses of different groups on day 28 after SCI. c Representative images from H&E staining in longitudinal section on day 28 after SCI. d , e The survived neurons following Nissl staining and relative quantification at the lesion area from different groups on day 28 after SCI. f The Basso, Beattie, and Bresnahan (BBB) locomotor scores of different groups. g The Immunofluorescence staining <t>of</t> <t>MAP2(green),</t> MBP (green), TUJ1 (red), and <t>GFAP</t> (red) from different groups on day 28 post-SCI. Data are presented as mean ± SD. * P < 0.05 vs. the SHAM group. # P < 0:05 vs. the SCI group. & P < 0.05 vs. the BMSC group ( n = 6)
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    The combined treatment of TUDCA and BMSC transplantation accelerated the regeneration of tissues and motor functional recovery after SCI. a Images of behavioral observation showing the characteristics of hind limb movement of rats across different groups. b Footprint analyses of different groups on day 28 after SCI. c Representative images from H&E staining in longitudinal section on day 28 after SCI. d , e The survived neurons following Nissl staining and relative quantification at the lesion area from different groups on day 28 after SCI. f The Basso, Beattie, and Bresnahan (BBB) locomotor scores of different groups. g The Immunofluorescence staining <t>of</t> <t>MAP2(green),</t> MBP (green), TUJ1 (red), and <t>GFAP</t> (red) from different groups on day 28 post-SCI. Data are presented as mean ± SD. * P < 0.05 vs. the SHAM group. # P < 0:05 vs. the SCI group. & P < 0.05 vs. the BMSC group ( n = 6)
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    Image Search Results


    The combined treatment of TUDCA and BMSC transplantation accelerated the regeneration of tissues and motor functional recovery after SCI. a Images of behavioral observation showing the characteristics of hind limb movement of rats across different groups. b Footprint analyses of different groups on day 28 after SCI. c Representative images from H&E staining in longitudinal section on day 28 after SCI. d , e The survived neurons following Nissl staining and relative quantification at the lesion area from different groups on day 28 after SCI. f The Basso, Beattie, and Bresnahan (BBB) locomotor scores of different groups. g The Immunofluorescence staining of MAP2(green), MBP (green), TUJ1 (red), and GFAP (red) from different groups on day 28 post-SCI. Data are presented as mean ± SD. * P < 0.05 vs. the SHAM group. # P < 0:05 vs. the SCI group. & P < 0.05 vs. the BMSC group ( n = 6)

    Journal: Molecular Neurobiology

    Article Title: Tauroursodeoxycholic Acid Inhibited Apoptosis and Oxidative Stress in H 2 O 2 -Induced BMSC Death via Modulating the Nrf-2 Signaling Pathway: the Therapeutic Implications in a Rat Model of Spinal Cord Injury

    doi: 10.1007/s12035-023-03754-5

    Figure Lengend Snippet: The combined treatment of TUDCA and BMSC transplantation accelerated the regeneration of tissues and motor functional recovery after SCI. a Images of behavioral observation showing the characteristics of hind limb movement of rats across different groups. b Footprint analyses of different groups on day 28 after SCI. c Representative images from H&E staining in longitudinal section on day 28 after SCI. d , e The survived neurons following Nissl staining and relative quantification at the lesion area from different groups on day 28 after SCI. f The Basso, Beattie, and Bresnahan (BBB) locomotor scores of different groups. g The Immunofluorescence staining of MAP2(green), MBP (green), TUJ1 (red), and GFAP (red) from different groups on day 28 post-SCI. Data are presented as mean ± SD. * P < 0.05 vs. the SHAM group. # P < 0:05 vs. the SCI group. & P < 0.05 vs. the BMSC group ( n = 6)

    Article Snippet: According to a previous study [ ], the tissues were subjected to overnight incubation at the temperature of 4 °C with microtubule-associated protein 2 (MAP2) (1:200, Boster Biological Engineering Co.), glial scar formation (GFAP) (1:200, Boster Biological Engineering Co.), myelin basic protein (MBP) (1:200, Boster Biological Engineering Co.), and beta-tubulin III (TUJ1) (1:200, CST) as primary antibodies.

    Techniques: Transplantation Assay, Functional Assay, Staining, Quantitative Proteomics, Immunofluorescence